Sodium glucose transporter 2 inhibition maintains kidney antibacterial response by decreasing complement C1q.
Kidney international
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Medical Faculty, University of Bonn University Hospital of Bonn Venusberg - Campus 1 53105 Bonn
In recent years, my group has addressed mechanisms of vascular inflammation in atherosclerosis and renal impairment, inflammatory mechanisms in peritoneal dialysis and in the kidney. Human cohort and sample (specifically renal biopsies, urine and peritoneal dialysate and tissue) collection have been central in both long-term (atherosclerosis, peritoneal dialysis) and acute clinical (EHEC-HUS) projects.
Kidney international
View this publicationPhysiological reports
View this publicationScience translational medicine
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