Pressure Overload-Induced Ventricular Crosstalk Activates Regenerative Mechanisms in the Contralateral Ventricle in Neonatal Mice.
Circulation
View this publicationMember
Medical Faculty, University of Bonn, University Hospital Bonn Sigmund-Freud-Str. 25 D-53105 Bonn
Circulation
View this publicationNature communications
View this publicationStem cell research
View this publication